When Is A Vaccine Injury Not An Injury? by
Wayne Rohdefor The Highwire
For most vaccines that are part of ‘the Program’, i.e. the National Vaccine Injury Compensation Program (NVICP), there is a list of known injuries and associated timeframes the injuries must manifest themselves within.
Very popular today is shoulder injury, as a result of a vaccine administration. The petition must show the pain and injury occurred within 48 hours to be considered a petition worthy of compensation by our government. But what about those petitioners that did not report the injury or document the injury until several days later.
For the NVICP to grant entitlement for compensation for a Guillain-Barré syndrome (GBS) case, the injury cannot occur prior to three days or no later than 42 days.
How did those dates and time frames become the standard to adjudicate petitions?
In the case of M. v HHS, petitioner alleges the meningococcal vaccine given to the child caused a neurologic condition known as chronic inflammatory demyelinating polyneuropathy (CIDP). Basically, severe chronic GBS.
The issue with this case is that the medical literature generally states that GBS and CIDP symptoms should not manifest themselves until the 3rd day from vaccination.
In this specific case, symptoms occurred within hours. Medical experts retained by the petitioner could not explain why or how symptoms occurred within hours. Case dismissed.
Yet the government’s position is that since the petitioner failed to establish a medical theory supported by research or medical expert testimony, the petitioner has failed on their burden to provide preponderance of evidence.
The Serotonin-Depression Myth & Facing The Culture That Promoted It by
Jefferey Jaxenfor The Highwire
The headlines have rung like a bell through popular culture and a public with fleeting trust in a medical-industrial-complex. A once sacred cow, settled science in the industry was that depression’s cause was a brain chemical imbalance problem. The chemical?….Serotonin – and Big Pharma’s decades long answer was a class of chemicals called Selective Serotonin Reuptake Inhibitors (SSRIs)…[but] Pump the breaks… hard.
Just published in the Journal Molecular Psychiatry, The serotonin theory of depression: a systematic umbrella review of the evidence looks at the principal relevant areas of research on the still influential serotonin hypothesis of depression.
The researchers write,
“The main areas of serotonin research provide no consistent evidence of there being an association between serotonin and depression, and no support for the hypothesis that depression is caused by lowered serotonin activity or concentrations. Some evidence was consistent with the possibility that long-term antidepressant use reduces serotonin concentration.”
The authors conclude:
“Our comprehensive review of the major strands of research on serotonin shows there is no convincing evidence that depression is associated with, or caused by, lower serotonin concentrations or activity.”
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